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Armod (Armodafinil)

Although results are equivocal so far, modafinil and Armod may also improve quality of life and global clinical condition in patients with OSA and residual sleepiness treated with CPAP therapy. No studies have examined whether Armod improves cognition in patients with MS.

Other names for this medication:
Armodafinil, Waklert, Armod, Nuvigil,

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Armod is used to treat excessive sleepiness caused by narcolepsy (a condition that causes excessive daytime sleepiness) or shift work sleep disorder (sleepiness during scheduled waking hours and difficulty falling asleep or staying asleep during scheduled sleeping hours in people who work at night or on rotating shifts). Armod is also used along with breathing devices or other treatments to prevent excessive sleepiness caused by obstructive sleep apnea/hypopnea syndrome (OSAHS; a sleep disorder in which the patient briefly stops breathing or breathes shallowly many times during sleep and therefore does not get enough restful sleep). Armod is in a class of medications called wakefulness-promoting agents. It works by changing the amounts of certain natural substances in the area of the brain that controls sleep and wakefulness.

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Given the medicinal relevance of the cardenolides in the treatment of congestive heart failure, a variety of ouabagenin analogs could potentially be generated from the key intermediate as a means of addressing the narrow therapeutic index of these molecules. The C2 domain protein Cts1 functions in the calcineurin signaling circuit during high-temperature stress responses in Cryptococcus neoformans. The bone volume/tissue volume ratio, trabecular thickness, trabecular separation, and trabecular number were evaluated for the two regions.

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Recent experiments have identified peptides that adhere to GaAs and Si surfaces. PLP mRNA was undetectable at Day 18 postfertilization but present in the white matter by Postnatal Day 0 (P0). Although the biologic function of ARID1A in breast cancer remains unknown, low expression of ARID1A can provide valuable prognostic information. non-MVC) during a 9-month period (January 2006 to September 2006).

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MicroRNA-99a/100 promotes apoptosis by targeting mTOR in human esophageal squamous cell carcinoma. Our results demonstrate that dasatinib, but not EGFR-directed treatment, can decrease cell viability of stromal cancer stem cells less sensitive to doxorubicin.

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Intra-operative formation of a thrombus in the right heart is rare and might be unrecognized. This may contribute to mitochondrial respiratory dysfunction during the pathogenesis of heart failure.

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A region spanning residues 509 to 673 reacted with most MAbs tested. Fashion and beauty products are displayed and informational materials provided. Clinically used antiepileptic drugs (AEDs) decrease membrane excitability by interacting with ion channels or neurotransmitter receptors. Osteoporotic fracture carries an enormous public health burden in terms of mortality and morbidity.

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